@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix schem: . @prefix go: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: species:9606; rdf:object go:0006954; rdf:predicate belv:increases; rdf:subject schem:12-Hydroxy-5%2C8%2C14-eicosatrienoic%20acid; a rdf:Statement . sub:assertion rdfs:label "a(SCHEM:\"12-Hydroxy-5,8,14-eicosatrienoic acid\") -> bp(GO:\"inflammatory response\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "1.4" . sub:_2 prov:value "CYP4B1 is induced by peroxisomal proliferators such as the hypolipidemic drug and clofibric acid, and it can metabolize fatty acids at the omega-carbon (Zeldin et al., 1995). 12-hydroxy-5,8,11, 14-eicosatetraenoic acid (12(R)-HETE) and 12-hydroxy-5,8, 14-eicosatrienoic acid (12(R)-HETrE ). Both metabolites possess potent inflammatory properties, with 12(R)-HETrE being a powerful angiogenic factor, and they assume the role of inflammatory mediators"; prov:wasQuotedFrom pubmed:10336559 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:10336559; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:29:47.221+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }