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All Rights Reserved. http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Small Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/dc/terms/license Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RA6FX_DZosU5vn28cuXGPxX64JXb9jGl_29S3mO3OxXVM#_7 http://resource.belframework.org/belframework/20131211/knowledge/small_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RA6FX_DZosU5vn28cuXGPxX64JXb9jGl_29S3mO3OxXVM#_6 http://www.w3.org/ns/prov#value To test whether the cell proliferation defect observed with Rin1 depletion was because of altered EGFR signaling, A549 cells were transfected with control or Rin1-specific morpholinos, serum-starved, and then stimulated with EGF. The levels of phosphorylated ERK1/2 (pERK) were used to assess EGF signaling (Fig. 2C). 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