@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix schem: .
@prefix go: .
@prefix species: .
@prefix occursIn: .
@prefix mesh: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 occursIn: mesh:D008099, mesh:D009132, species:9606;
rdf:object go:0006094;
rdf:predicate belv:decreases;
rdf:subject schem:serum%20insulin;
a rdf:Statement .
sub:assertion rdfs:label "a(SCHEM:\"serum insulin\") -| bp(GO:gluconeogenesis)" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_3;
pav:version "1.4" .
sub:_2 prov:value "Both obesity and lipoatrophy also cause insulin resistance and predisposition to type 2 diabetes, demonstrating that adipose tissue is crucial in regulating metabolism beyond its ability to take up glucose4. Although insulin does not stimulate glucose uptake in liver, it blocks glycogenolysis and gluconeogenesis, and stimulates glycogen synthesis, thus regulating fasting glucose levels.";
prov:wasQuotedFrom pubmed:11742412 .
sub:_3 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:11742412;
prov:wasDerivedFrom beldoc:, sub:_2 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:29:59.628+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}