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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RA9ypolyq4z-YMXPEIX9LliI7SN-C1eieq8TwQGsDrq7k#_9 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RA9ypolyq4z-YMXPEIX9LliI7SN-C1eieq8TwQGsDrq7k#_8 http://www.w3.org/ns/prov#value In vivo, endogenous FAP68 can be coimmunoprecipitated with the HGF receptor in the absence of stimuli and not upon HGF stimulation. Thus, FAP68 represents a novel type of effector that interacts with the inactive HGF receptor and is released upon receptor phosphorylation. # Ariadne: Significantly, nonphosphorylated HGF receptor prevents FAP68 from stimulating p70S6K . [Regulation] # Ariadne: FAP68 interacts specifically with the inactive form of HGF receptor, such as a kinase- defective receptor or a dephosphorylated wild type receptor. [Binding] # Ariadne: Significantly, nonphosphorylated HGF receptor prevents FAP68 from stimulating p70S6K . 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