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[These results show that the loss of Atp13a2 causes sensorimotor impairments, ?-synuclein accumulation as occurs in PD and related synucleinopathies, and accumulation of lipofuscin deposits characteristic of NCL, thus providing the first direct demonstration that null mutations in Atp13a2 can cause pathological features of both diseases in the same organism.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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