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http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc
http://www.nanopub.org/nschema#hasProvenance
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http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc
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http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#assertion
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#_1
http://www.selventa.com/vocabulary/variantOf
http://rgd.mcw.edu/rgdweb/report/gene/main.html?id=3772
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000
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http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00696
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#_2
http://purl.obolibrary.org/obo/BFO_0000066
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http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#_2
http://www.w3.org/1999/02/22-rdf-syntax-ns#predicate
http://www.selventa.com/vocabulary/increases
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http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://resource.belframework.org/belframework/1.0/namespace/selventa-legacy-chemical-names/serum%20leptin
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http://www.w3.org/2000/01/rdf-schema#label
a(SCHEM:"serum leptin") -> p(RGD:Stat3,pmod(P))
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://purl.org/pav/version
1.4
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http://www.w3.org/ns/prov#value
In trh promoter assays using transfected cells, functional STAT3 proteins are required for maximal activation of the trh promoter by leptin. Consistent with this, the STAT3-binding site on the leptin receptor is also required for this regulation. Using double immunohistochemistry, we show that peripherally administered leptin rapidly stimulates STAT3 phosphorylation in approximately 40% of TRH neurons in the paraventricular nucleus of the hypothalamus (PVN) in rats. Detailed anatomical analyses reveal that the leptin-responsive TRH neurons are concentrated in the caudal region of the medial and periventricular parvocellular subnucleus of the PVN. Combined, our data show that only a subpopulation of TRH neurons in the PVN is leptin responsive and suggest that stimulation of hypothalamic trh gene expression by leptin involves activation of STAT3 and that this signaling pathway is important for regulation of the hypothalamic-pituitary-thyroid axis by leptin.
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#_3
http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/14764629
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.ncbi.nlm.nih.gov/pubmed/14764629
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#_3
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc#pubinfo
http://www.tkuhn.ch/bel2nanopub/RAQtYDo5NaHsCD00_BXVWwi8GrMfhcKQtYFBAG7FbaOZc
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2014-07-03T14:30:20.322+02:00
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http://orcid.org/0000-0002-1267-0234