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[Importantly, activated Notch1, Notch2, and HES1 all led to inhibited AML growth in vivo, and Notch inhibition via dnMAML enhanced proliferation in vivo, thus revealing the physiological inhibition of AML growth in vivo in response to Notch signaling.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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