Nanopublications

sub:assertion {
  sub:_1 hasAgent: sub:_2 ;
    a go:0016301 .
  sub:_2 geneProductOf: hgnc:6876 ;
    a Protein: .
  sub:_3 hasAgent: sub:_4 ;
    a go:0042789 .
  sub:_4 geneProductOf: hgnc:6993 ;
    a Protein: .
  sub:_5 rdf:object sub:_3 ;
    rdf:predicate belv:increases ;
    rdf:subject go:0006970 ;
    a rdf:Statement .
  sub:_6 occursIn: species:9606 ;
    rdf:object sub:_5 ;
    rdf:predicate belv:increases ;
    rdf:subject sub:_1 ;
    a rdf:Statement .
  sub:assertion rdfs:label "bp(GO:\"response to osmotic stress\") -> tscript(p(HGNC:MEF2A))" , "kin(p(HGNC:MAPK14)) -> (bp(GO:\"response to osmotic stress\") -> tscript(p(HGNC:MEF2A)))" .
}

sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_8 ;
    pav:version "1.4" .
  sub:_7 prov:value "All stimuli resulted primarily in activation of MEF2D DNA binding. Exposure of cells to osmotic or oxidative stress increased MEF2 DNA binding via pathways that were completely blocked by MAPK inhibitors and partially blocked by inhibitors of PKC, PI 3-kinase, and AMPK." ;
    prov:wasQuotedFrom pubmed:14960415 .
  sub:_8 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:14960415 ;
    prov:wasDerivedFrom beldoc: , sub:_7 .
}