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http://www.nanopub.org/nschema#hasPublicationInfo
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#_1
http://purl.obolibrary.org/obo/BFO_0000066
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http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://amigo.geneontology.org/amigo/term/GO:0007049
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#predicate
http://www.selventa.com/vocabulary/increases
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://amigo.geneontology.org/amigo/term/GO:0009314
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#assertion
http://www.w3.org/2000/01/rdf-schema#label
bp(GO:"response to radiation") -> bp(GO:"cell cycle")
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#provenance
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
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http://purl.org/dc/elements/1.1/rights
Copyright (c) 2011-2012, Selventa. All rights reserved.
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/title
BEL Framework Large Corpus Document
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1.4
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http://www.w3.org/ns/prov#value
Activation of the tumor suppressor p53 by DNA damage induces either cell cycle arrest or apoptosis, but what determines the choice between cytostasis and death is not clear. In this report, we show that the E1A-binding p300 nucleoprotein is a key determinant of p53-dependent cell fate in colorectal cancer cells: absence of p300 increases apoptosis in response to DNA damage. In addition, p300-deficient (p300(-)) cells fail to undergo G(1)/S arrest after UV irradiation. These abnormalities are associated with prolongation of p53 stability, reduced p53-acetylation, blunting of MDM2 activation, failure to transactivate p21, and a disproportionate increase in PUMA levels. When xenografted, p300(-) cells are more sensitive to chemotherapy with doxorubicin. These results show that p300 is a key regulator of the p53 response and suggest that p300 inhibition could be used to modulate chemotherapy.
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#_2
http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
http://www.ncbi.nlm.nih.gov/pubmed/15123817
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM#pubinfo
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM
http://purl.org/dc/terms/created
2014-07-03T14:30:23.175+02:00
http://www.tkuhn.ch/bel2nanopub/RASjoioybDgykCoAotp_kGwg7tsnvJvvDY3c1Vk6JvnkM
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http://orcid.org/0000-0001-6818-334X
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http://orcid.org/0000-0002-1267-0234