@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix go: . @prefix hasAgent: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:1582; a Protein: . sub:_2 hasAgent: sub:_3; a go:0042789 . sub:_3 geneProductOf: hgnc:7545; a Protein: . sub:_4 occursIn: species:9606; rdf:object sub:_2; rdf:predicate belv:directlyDecreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:CCND1) =| tscript(p(HGNC:MYB))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_6; pav:version "1.4" . sub:_5 prov:value "Previous studies have demonstrated a role for cyclin D1 in the repression of several distinct transcription factors (Fig. 4). In addition to binding CDK4, CDK6, and pRB, cyclin D1 forms physical associations with P/CAF (p300/CB P associated factor), Myb, MyoD, and the cyclin D1 myb-like binding protein (DMP1) [8-11]. Cyclin D1 and D2 are capable of inhibiting MyoD [12]. v-Myb is preferentially repressed by cyclin D1 [9], although cyclin D2 and D3 are also capable of repressing v-Myb; the helix-loop-helix-like protein DMP1 is repressed by each of the D-type cyclins [8]. In contrast, the repression of BETA2/NeuroD by cyclin D1 is not reconstituted by either cyclin D2 or D3."; prov:wasQuotedFrom pubmed:15320717 . sub:_6 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:15320717; prov:wasDerivedFrom beldoc:, sub:_5 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:27.089+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }