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p(HGNC:MCL1,pmod(P,S,121)) -> bp(MESHPP:Apoptosis)
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Approximately 61,000 statements.
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We found that JNK phosphorylated Ser-121 and Thr-163 of Mcl-1 in response to stimulation with H(2)O(2) and that transfection of unphosphorylatable Mcl-1 resulted in an enhanced anti-apoptotic activity in response to stimulation with H(2)O(2).
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