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supplementation with RXR attenuated these inhibitions by Ppars implicating receptor interaction among LXR, Ppar and RXR as a plausible mechanism Pparalpha activation can suppress LXR-Srebp1c pathway through reduction of LXR/RXR formation, proposing a novel transcription factor cross-talk between LXR and Pparalpha in hepatic lipid homeostasis
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