@prefix this: <http://www.tkuhn.ch/bel2nanopub/RAWR-JAgoiAYHQpF1L3y6rtgo3FGDGC1ehIb3Ztaby9xE> .
@prefix sub: <http://www.tkuhn.ch/bel2nanopub/RAWR-JAgoiAYHQpF1L3y6rtgo3FGDGC1ehIb3Ztaby9xE#> .
@prefix beldoc: <http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix rdf: <http://www.w3.org/1999/02/22-rdf-syntax-ns#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix dct: <http://purl.org/dc/terms/> .
@prefix dce: <http://purl.org/dc/elements/1.1/> .
@prefix pav: <http://purl.org/pav/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix belv: <http://www.selventa.com/vocabulary/> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix mesh: <http://purl.bioontology.org/ontology/MSH/> .
@prefix schem: <http://resource.belframework.org/belframework/1.0/namespace/selventa-legacy-chemical-names/> .
@prefix species: <http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=> .
@prefix occursIn: <http://purl.obolibrary.org/obo/BFO_0000066> .
@prefix pubmed: <http://www.ncbi.nlm.nih.gov/pubmed/> .
@prefix orcid: <http://orcid.org/> .
sub:Head {
  this: np:hasAssertion sub:assertion ;
    np:hasProvenance sub:provenance ;
    np:hasPublicationInfo sub:pubinfo ;
    a np:Nanopublication .
}
sub:assertion {
  sub:_1 occursIn: mesh:D018482 , species:9606 ;
    rdf:object schem:serum%20glucose ;
    rdf:predicate belv:positiveCorrelation ;
    rdf:subject mesh:D007333 ;
    a rdf:Statement .
  sub:assertion rdfs:label "bp(MESHPP:\"Insulin Resistance\") positiveCorrelation a(SCHEM:\"serum glucose\")" .
}
sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_3 ;
    pav:version "1.4" .
  sub:_2 prov:value "GLUT4 has a crucial role in whole-body glucose homeostasis insulin-stimulated glucose transport is an important rate-limiting step for glucose metabolism in both muscle and fat tissue, and is severely disrupted in type II diabetes The disease (type II diabetes) is characterized by defective insulin action, a condition that is referred to as insulin resistance. Insulin resistance is characterized by dysfunctional glucose uptake into muscle and adipose tissue, in conjunction with an oversupply of glucose from the liver, which results in high circulating glucose levels." ;
    prov:wasQuotedFrom pubmed:11994746 .
  sub:_3 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:11994746 ;
    prov:wasDerivedFrom beldoc: , sub:_2 .
}
sub:pubinfo {
  this: dct:created "2014-07-03T14:30:02.934+02:00"^^xsd:dateTime ;
    pav:createdBy orcid:0000-0001-6818-334X , orcid:0000-0002-1267-0234 .
}