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http://www.nanopub.org/nschema#hasPublicationInfo
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAWR-JAgoiAYHQpF1L3y6rtgo3FGDGC1ehIb3Ztaby9xE#_1
http://purl.obolibrary.org/obo/BFO_0000066
http://purl.bioontology.org/ontology/MSH/D018482
http://www.tkuhn.ch/bel2nanopub/RAWR-JAgoiAYHQpF1L3y6rtgo3FGDGC1ehIb3Ztaby9xE#_1
http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
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http://www.w3.org/1999/02/22-rdf-syntax-ns#predicate
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http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.bioontology.org/ontology/MSH/D007333
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.w3.org/1999/02/22-rdf-syntax-ns#Statement
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http://www.w3.org/2000/01/rdf-schema#label
bp(MESHPP:"Insulin Resistance") positiveCorrelation a(SCHEM:"serum glucose")
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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GLUT4 has a crucial role in whole-body glucose homeostasis insulin-stimulated glucose transport is an important rate-limiting step for glucose metabolism in both muscle and fat tissue, and is severely disrupted in type II diabetes The disease (type II diabetes) is characterized by defective insulin action, a condition that is referred to as insulin resistance. Insulin resistance is characterized by dysfunctional glucose uptake into muscle and adipose tissue, in conjunction with an oversupply of glucose from the liver, which results in high circulating glucose levels.
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http://www.w3.org/ns/prov#wasQuotedFrom
http://www.ncbi.nlm.nih.gov/pubmed/11994746
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://purl.org/dc/terms/created
2014-07-03T14:30:02.934+02:00
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