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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAWSQ_hYcQ1OaUt0Bzs2eHMnXWmAcnndnBeatdLzQuqvs#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RAWSQ_hYcQ1OaUt0Bzs2eHMnXWmAcnndnBeatdLzQuqvs#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0016301
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http://purl.obolibrary.org/obo/RO_0002204
http://www.informatics.jax.org/marker/MGI:1346865
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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kin(p(MGI:Mapk14)) -> p(MGI:Hnrnpa1,pmod(P))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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We show that exposure of NIH-3T3 and SV-40 transformed green monkey kidney (COS) cells to stress stimuli such as osmotic shock or UVC irradiation, but not to mitogenic activators such as PDGF or EGF, results in a marked cytoplasmic accumulation of hnRNP A1, concomitant with an increase in its phosphorylation. These effects are mediated by the MKK(3/6)-p38 pathway, and moreover, p38 activation is necessary and sufficient for the induction of hnRNP A1 cytoplasmic accumulation.
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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2014-07-03T14:29:50.742+02:00
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