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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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In addition, we (12) and others (13,14) have previously demonstrated that TNF-A represses the mRNA levels of an adipocyte-derived hormone, ACRP30, which inhibits gluconeogenesis in the liver (15), and decreases plasma levels of glucose (16) and FFAs (17). Thus, the increased circulating FFA concentrations and/or reduced ACRP30 protein levels could mediate some of the delayed effects of TNF-A on whole-body energy metabolism.
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2014-07-03T14:30:06.725+02:00
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