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http://www.tkuhn.ch/bel2nanopub/RAYxEl9kIhGFyfkSy6QMDVAdhmPKrMpAvbIpjoTxbPkuE#_1
http://semanticscience.org/resource/SIO_000139
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http://purl.obolibrary.org/obo/RO_0002204
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://www.selventa.com/vocabulary/variantOf
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kin(p(HGNC:PRKCZ)) => p(HGNC:YWHAB,pmod(P))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Together, the results suggest that 14-3-3 binds both PKC-zeta (at phospho-Ser-186) and Raf-1 in a ternary complex. Phosphorylation of 14-3-3beta by PKC-zeta negatively regulated their physical association. 14-3-3beta with its putative PKC-zeta phosphorylation sites mutated enhanced co-precipitation between PKC-zeta and Raf-1, suggesting that phosphorylation of 14-3-3 by PKC-zeta weakens the complex in vivo. We conclude that 14-3-3 facilitates coupling of PKC-zeta to Raf-1 in an isotype-specific and phosphorylation-dependent manner. We suggest that 14-3-3 is a transient mediator of Raf-1 phosphorylation and activation by PKC-zeta.
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Selventa
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2014-07-03T14:29:49.584+02:00
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