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[Along with SM22? induction in PHN, confirmed at both mRNA and protein levels, SM22? was also induced across a broad range of proteinuric diseases, including experimental animal models (puromycin aminonucleoside nephropathy, adriamycin nephropathy, passive nephrotoxic nephritis, and diet-induced obesity) and human diseases (collapsing glomerulopathy, diabetic nephropathy, classic focal segmental glomerulosclerosis, IgA nephropathy, minimal-change disease, membranous nephropathy, and membranoproliferative glomerulonephritis).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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