@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix go: . @prefix species: . @prefix occursIn: . @prefix mesh: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: mesh:D042783, species:9606; rdf:object go:0072672; rdf:predicate belv:increases; rdf:subject chebi:26523; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:\"reactive oxygen species\") -> bp(GO:\"neutrophil extravasation\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "1.4" . sub:_2 prov:value "some of these factors also initiate the conversion from the XD to the XO form [5 and 6]. Endothelial XOR was suggested to trigger a microvascular inflammatory response, leading to recruitment and activation of circulating neutrophils and trapping of circulating pathogens [6]. The recruitment of neutrophils and the activation of macrophages are induced by ROS that upregulate cell adhesion molecules [40 and 41]."; prov:wasQuotedFrom pubmed:12967676 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12967676; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:16.448+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }