sub:provenance {
  beldoc: dce:description "Approximately 2000 hand curated statements drawn from 57 PubMeds." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All Rights Reserved." ;
    dce:title "BEL Framework Small Corpus Document" ;
    dc:license "Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License" ;
    pav:authoredBy sub:_6 ;
    pav:version "1.6" .
  sub:_5 prov:value """In contrast, tissues treated with TNFR1 antibody, although hyperplastic, retained
BMZ structure and proper desmoglein-3 expression, indicating intact tissue polarity
(Fig. 5A and B). Blockade of TNFR1 signaling was accompanied by the absence
of detectable pJNK in the tissues treated by the TNFR1 antibody (Supplementary
Fig. S7). In addition, TNFR1 blockade reduced the mitotic index and limited mitotic
cells to the basal layer, whereas proliferating cells were widespread and abundant
in the control tumor tissue (Fig. 5C and D). These findings indicate that
antagonizing TNFR1 function prevents Ras-driven tumorigenesis.""" ;
    prov:wasQuotedFrom pubmed:17440097 .
  sub:_6 rdfs:comment "support@belframework.org" ;
    rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:17440097 ;
    prov:wasDerivedFrom beldoc: , sub:_5 .
}