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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0043234
http://www.tkuhn.ch/bel2nanopub/RAaZlDo7uwbx25r4lcVFwUV33uKqDDLPHX5M0b2ao6uJE#_1
http://semanticscience.org/resource/SIO_000139
http://resource.belframework.org/belframework/1.0/namespace/selventa-named-human-complexes/p85%2Fp110%20PI3Kinase%20Complex
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0016301
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http://purl.obolibrary.org/obo/RO_0002204
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http://www.w3.org/2000/01/rdf-schema#label
kin(complex(NCH:"p85/p110 PI3Kinase Complex")) -> kin(p(HGNC:AKT1))
http://www.tkuhn.ch/bel2nanopub/RAaZlDo7uwbx25r4lcVFwUV33uKqDDLPHX5M0b2ao6uJE#provenance
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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Overexpressed IRS-3 as well as IRS-1 enhanced phosphoinositide (PI) 3-kinase activity in response to insulin and increased phosphorylation of protein kinase B (PKB) at S473 and phosphorylation of one of the members of the forkhead transcription factor FKHRL1 on T32 in both insulin-untreated and -treated states.
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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2014-07-03T14:30:08.984+02:00
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