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Approximately 61,000 statements. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/rights Copyright (c) 2011-2012, Selventa. All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAaaAW5vuE2hEElstl90r06sibT9dhT_4eb_p1wXy-CtQ#_8 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAaaAW5vuE2hEElstl90r06sibT9dhT_4eb_p1wXy-CtQ#_7 http://www.w3.org/ns/prov#value Retinoids, or vitamin A analogs, are among the most promising chemopreventive agents for breast cancer (Table I). Two classes of retinoid receptors, RARand RXR, bind retinoids and mediate transcriptional regulation of genes that control cellular proliferation, differentiation, and apoptosis (52). RXR is able to interact with many other factors such as vitamin D receptor (VDR), peroxisome proliferator- activator receptor (PPAR), thyroid hormone receptor, and COUP (Fig. 1). 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