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http://www.tkuhn.ch/bel2nanopub/RAbaX8cZXNlLCAncU2CVtcTRYncAWWFEjB6_MHfUoJCYg
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RAbaX8cZXNlLCAncU2CVtcTRYncAWWFEjB6_MHfUoJCYg#_1
http://purl.obolibrary.org/obo/RO_0002204
http://resource.belframework.org/belframework/1.0/namespace/selventa-named-human-protein-families/VEGF%20Family
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/RO_0002204
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http://www.selventa.com/vocabulary/increases
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http://www.w3.org/2000/01/rdf-schema#label
p(PFH:"VEGF Family") -> r(HGNC:CXCR4)
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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BEL Framework Large Corpus Document
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1.4
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In fact, our studies indicate that VEGF/neuropilin-1 autocrine signaling promotes the migration of breast carcinoma cells by increasing the expression of the chemokine receptor CXCR4 [19]. Our finding that VEGF regulates CXCR4 expression is relevant because stromal-derived factor-1, the ligand for this receptor, is present in tumor stroma and in tissues such as lymph and lung [36], which are the primary targets of invasive breast carcinoma cells, and CXCR4 inhibitors impair metastasis.
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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http://purl.org/dc/terms/created
2014-07-03T14:30:21.365+02:00
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