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http://www.tkuhn.ch/bel2nanopub/RAcUAyWP2D9aregPFcx96RTsMvAqye9t7LR2NOrszzhBY#_1
http://semanticscience.org/resource/SIO_000139
http://www.tkuhn.ch/bel2nanopub/RAcUAyWP2D9aregPFcx96RTsMvAqye9t7LR2NOrszzhBY#_2
http://www.tkuhn.ch/bel2nanopub/RAcUAyWP2D9aregPFcx96RTsMvAqye9t7LR2NOrszzhBY#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0003824
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
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cat(p(MGI:Casp3)) -> bp(MESHPP:Apoptosis)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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http://www.w3.org/ns/prov#value
KO mice showed a significantly greater infarction volume and DNA fragmentation in the cortex than WT mice at 24 hours after ischemia. At 8 hours, cytochrome c release into the cytoplasm was markedly higher in KO mice than in WT mice. Caspase-3 activation was also significantly enhanced in KO mice versus WT mice the deletion of the hsp70.1 gene increases cytochrome c release into the cytoplasm and subsequent caspase-3 activation, thereby exacerbating apoptosis after focal cerebral ischemia.
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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2014-07-03T14:30:25.760+02:00
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