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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAchc54GC6H6aPb2nhwgqrxuoe88_Fwn63fTY1hEbSs70#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RAchc54GC6H6aPb2nhwgqrxuoe88_Fwn63fTY1hEbSs70#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0042789
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=5021
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/2000/01/rdf-schema#label
tscript(p(HGNC:FOXA1)) => r(HGNC:CDKN1B)
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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http://www.w3.org/ns/prov#value
In summary, we identified a FOXA1 binding site within the BRCA1-responsive element of the p27(Kip1) promoter and showed that FOXA1 activated the promoter alone and in conjunction with BRCA1. Furthermore, we identified high expression of FOXA1 in breast cancer cell lines and tissues, discovered a role for BRCA1 in the regulation of p27(Kip1) transcription and a possible interaction with BRCA1.
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
http://www.tkuhn.ch/bel2nanopub/RAchc54GC6H6aPb2nhwgqrxuoe88_Fwn63fTY1hEbSs70#assertion
http://www.w3.org/ns/prov#wasDerivedFrom
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http://purl.org/dc/terms/created
2014-07-03T14:30:39.955+02:00
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http://orcid.org/0000-0001-6818-334X
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