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http://www.nanopub.org/nschema#hasAssertion
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http://www.nanopub.org/nschema#hasPublicationInfo
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http://www.tkuhn.ch/bel2nanopub/RAdZWydUWmSpxPEzACLjevJsP7DJvm7Qy6aR-6PFpT2gQ
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RAdZWydUWmSpxPEzACLjevJsP7DJvm7Qy6aR-6PFpT2gQ#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=3236
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/BFO_0000066
http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=9606
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http://www.w3.org/2000/01/rdf-schema#label
p(HGNC:EGFR) -> p(HGNC:ANXA1,pmod(P,Y,21))
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http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://purl.org/pav/authoredBy
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http://purl.org/pav/version
1.4
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http://www.w3.org/ns/prov#value
Deletion of annexin 1, a substrate of EGFR tyrosine kinase, abolishes the effect of EGF stimulation on inward vesiculation. This phenotype is reversible by transfection with wild-type but not Y21F phosphorylation mutant annexin 1.
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RAdZWydUWmSpxPEzACLjevJsP7DJvm7Qy6aR-6PFpT2gQ
http://purl.org/dc/terms/created
2014-07-03T14:30:37.029+02:00
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