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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAe7qN8v8wIvODbaaS5PkqHNJu83cLWfdfmtSwg0n-eYs#_4 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAe7qN8v8wIvODbaaS5PkqHNJu83cLWfdfmtSwg0n-eYs#_3 http://www.w3.org/ns/prov#value Additional evidence of the involvement of these inflammatory pathways is supported by the protective effect of some anti-inflammatory drugs against obesity-induced insulin resistance. Aspirin may inhibit not only IKK and JNK (93,94) but other Ser/Thr kinases (mamalian target of rapamycin and protein kinase B/Akt) related to TNF-alpha insulin resistance by phosphorylation of IRS-1 at serine residues (94). 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