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http://www.nanopub.org/nschema#hasProvenance
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAeHZgIyhpD0yYAjA23ycF5xR7tgRsjb_t9pNc2_WfJLY#assertion
http://www.tkuhn.ch/bel2nanopub/RAeHZgIyhpD0yYAjA23ycF5xR7tgRsjb_t9pNc2_WfJLY#_1
http://purl.obolibrary.org/obo/RO_0002204
http://rgd.mcw.edu/rgdweb/report/gene/main.html?id=2868
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http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://amigo.geneontology.org/amigo/term/GO:0014734
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http://www.w3.org/1999/02/22-rdf-syntax-ns#predicate
http://www.selventa.com/vocabulary/increases
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http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
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http://www.w3.org/2000/01/rdf-schema#label
p(RGD:Igf1) -> bp(GO:"skeletal muscle hypertrophy")
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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However, the absence of a hypertrophy response to IGF-I may indicate that the presence of increased IGFBP-4 levels reduced ligation of the IGF-I receptor, therefore limiting anabolic processes. This result would be in accord with the lesser increase in S6K1 phosphorylation seen with PD coinfusion
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RAeHZgIyhpD0yYAjA23ycF5xR7tgRsjb_t9pNc2_WfJLY#pubinfo
http://www.tkuhn.ch/bel2nanopub/RAeHZgIyhpD0yYAjA23ycF5xR7tgRsjb_t9pNc2_WfJLY
http://purl.org/dc/terms/created
2014-07-03T14:30:16.244+02:00
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http://orcid.org/0000-0001-6818-334X
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