@prefix this: <http://www.tkuhn.ch/bel2nanopub/RAeosPKZ7UFj-U64_mbdwnsBXhzfDPuFp0RBKJ_5QhFZg> .
@prefix sub: <http://www.tkuhn.ch/bel2nanopub/RAeosPKZ7UFj-U64_mbdwnsBXhzfDPuFp0RBKJ_5QhFZg#> .
@prefix beldoc: <http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix rdf: <http://www.w3.org/1999/02/22-rdf-syntax-ns#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix dct: <http://purl.org/dc/terms/> .
@prefix dce: <http://purl.org/dc/elements/1.1/> .
@prefix pav: <http://purl.org/pav/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix belv: <http://www.selventa.com/vocabulary/> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix Protein: <http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080> .
@prefix hgnc: <http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=> .
@prefix geneProductOf: <http://purl.obolibrary.org/obo/RO_0002204> .
@prefix species: <http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=> .
@prefix occursIn: <http://purl.obolibrary.org/obo/BFO_0000066> .
@prefix mesh: <http://purl.bioontology.org/ontology/MSH/> .
@prefix pubmed: <http://www.ncbi.nlm.nih.gov/pubmed/> .
@prefix orcid: <http://orcid.org/> .
sub:Head {
  this: np:hasAssertion sub:assertion ;
    np:hasProvenance sub:provenance ;
    np:hasPublicationInfo sub:pubinfo ;
    a np:Nanopublication .
}
sub:assertion {
  sub:_1 geneProductOf: hgnc:11766 ;
    a Protein: .
  sub:_2 geneProductOf: hgnc:336 ;
    a Protein: .
  sub:_3 occursIn: mesh:D008168 , species:9606 ;
    rdf:object sub:_2 ;
    rdf:predicate belv:increases ;
    rdf:subject sub:_1 ;
    a rdf:Statement .
  sub:assertion rdfs:label "p(HGNC:TGFB1) -> p(HGNC:AGTR1)" .
}
sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_5 ;
    pav:version "1.4" .
  sub:_4 prov:value "TGF-1beta treatment significantly decreased miR-155 expression levels (Fig. 6A). Although TGF-1beta also up-regulated the expression of the hAT1R gene (Fig. 6B), the time course for the increased production of hAT1R mRNA did not correlate well with the time course for the enhanced synthesis of hAT1R protein levels since these values remained higher than expected at the 8 and 12 h time points (Fig. 6, C versus B). Therefore, the sustained increase in hAT1R protein levels cannot be accounted for by only a transcriptional mechanism and suggests that post-transcriptional mechanisms must also be involved." ;
    prov:wasQuotedFrom pubmed:16675453 .
  sub:_5 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:16675453 ;
    prov:wasDerivedFrom beldoc: , sub:_4 .
}
sub:pubinfo {
  this: dct:created "2014-07-03T14:30:42.944+02:00"^^xsd:dateTime ;
    pav:createdBy orcid:0000-0001-6818-334X , orcid:0000-0002-1267-0234 .
}