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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAexvqH1BrH2mRIqaHKUg_79D8psDdEF2cHg4e61fOAFo#_1
http://semanticscience.org/resource/SIO_000139
http://www.tkuhn.ch/bel2nanopub/RAexvqH1BrH2mRIqaHKUg_79D8psDdEF2cHg4e61fOAFo#_2
http://www.tkuhn.ch/bel2nanopub/RAexvqH1BrH2mRIqaHKUg_79D8psDdEF2cHg4e61fOAFo#_1
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http://amigo.geneontology.org/amigo/term/GO:0016301
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
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http://www.w3.org/2000/01/rdf-schema#label
kin(p(PFH:"PRKC Family")) -> r(HGNC:PLAU)
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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http://www.w3.org/ns/prov#value
Increased expression of PKC in malignant and aggressive breast cancers was recently demonstrated, and PKC activity has been shown to be higher in breast cancers than in normal breast tissue [68, 69]. In addition, high levels of PKC? and -? were correlated with enhanced uPA secretion in highly invasive MDA-MB- 231 breast cancer cells [70]. Furthermore, the introduction of PKC? gene to nonmetastatic MCF-7 breast cancer cells resulted in more aggressive neoplastic phenotype, and the stimulation of PKC activity with phorbol ester, phorbol-12- myristate-13-acetate (PMA), resulted in increases in both invasiveness and uPA expression
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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2014-07-03T14:30:23.975+02:00
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