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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAg10DUKW-E9UUGQim7lqEfoFCf57URrN6J1odJ8kRYhs#_5 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAg10DUKW-E9UUGQim7lqEfoFCf57URrN6J1odJ8kRYhs#_4 http://www.w3.org/ns/prov#value Increased oxygen radical production may not only contribute to posttransplant ischemia-reperfusion injury but also to acute rejection of renal allografts. Xanthine oxidoreductase (XOR) may constitute a relevant reactive oxygen species (ROS) source. CONCLUSIONS: A major part of ROS generation in acute rejection was contributed by XOR. 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