@prefix dcterms: . @prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix go: . @prefix nch: . @prefix ProteinComplex: . @prefix hasAgent: . @prefix sdis: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { nch:Nfkb%20Complex a ProteinComplex: . sub:_1 hasAgent: nch:Nfkb%20Complex; a go:0042789 . sub:_2 occursIn: species:9606; rdf:object sdis:cardiomyocyte%20hypertrophy; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "tscript(complex(NCH:\"Nfkb Complex\")) -> path(SDIS:\"cardiomyocyte hypertrophy\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "1.4" . sub:_3 prov:value "Overexpression of a constitutively active mutant of ASK1 led to NF kappaB activation and cardiac hypertrophy. Activated ASK1-induced hypertrophy was abolished by inhibition of NF-kappaB activation"; prov:wasQuotedFrom pubmed:11815436 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:11815436; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dcterms:created "2014-07-03T14:30:00.743+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }