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[We used genetically modified, atherosclerosis-prone mice, including mice with macrophage-specific uPA overexpression and mice genetically deficient in uPAR to elucidate mechanisms of uPA/uPAR/plasminogen-accelerated atherosclerosis and aneurysm formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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