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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAgvEZGXW2z4-23HfUnjeHjzPakEhGX2SMTXHV0Auljiw#_4 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAgvEZGXW2z4-23HfUnjeHjzPakEhGX2SMTXHV0Auljiw#_3 http://www.w3.org/ns/prov#value TSA PROMOTES EXPRESSION OF FOLLISTATIN AND OF MARKERS OF MUSCLE REGENERATION We next wished to evaluate whether HDAC inhibitors may affect muscle regeneration in vivo. To this end, TSA was systemically delivered to adult female mice with subcutaneous micro-osmotic pumps. After 2 days of TSA treatment, the tibialis anterior muscles were in- jected with cardiotoxin. TSA delivery was continued for 3 additional days, after which the animals were sacrificed. At this time, muscle regeneration has been initi- ated (Charge and Rudnicki, 2004). The expression levels of follistatin in noninjured muscles was almost undetectable and increased upon injury (data not shown and Supplemental Figure S1). 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