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http://www.tkuhn.ch/bel2nanopub/RAhRxjqBicTMJyvA4lfWi4VOVUKMHjm3wg4sqrrW2AV0w
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAhRxjqBicTMJyvA4lfWi4VOVUKMHjm3wg4sqrrW2AV0w#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RAhRxjqBicTMJyvA4lfWi4VOVUKMHjm3wg4sqrrW2AV0w#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0003824
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http://purl.obolibrary.org/obo/RO_0002204
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http://purl.obolibrary.org/obo/BFO_0000066
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http://www.w3.org/2000/01/rdf-schema#label
a(CHEBI:"trichostatin A") =| cat(p(PFM:"Hdac Family"))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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http://www.w3.org/ns/prov#value
The expression of 10 histone deacetylases (HDAC1-10) mRNAs in mouse neuroblastoma Neuro-2a and microglia N9 cell cultures after treatment by inhibitors of HDACs, sodium butyrate and trichostatin A was studied to elucidate whether HDAC inhibitors affect the gene expression of HDACs In both Neuro-2a and N9 cells the highest increase was observed for HDAC5 and -6 mRNA, whereas, HDAC4 had a prominent increase in mRNA levels after drug treatment only in N9 microglia cell line but not in Neuro-2a.
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel
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http://www.tkuhn.ch/bel2nanopub/RAhRxjqBicTMJyvA4lfWi4VOVUKMHjm3wg4sqrrW2AV0w
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2014-07-03T14:30:25.608+02:00
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