sub:provenance {
  beldoc: dce:description "Approximately 2000 hand curated statements drawn from 57 PubMeds." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All Rights Reserved." ;
    dce:title "BEL Framework Small Corpus Document" ;
    dc:license "Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License" ;
    pav:authoredBy sub:_7 ;
    pav:version "1.6" .
  sub:_6 prov:value """In ECs, VEGF stimulates ARF6 association with Rac1, which is cotemporaneous with Rac1
activation. The dominant negative ARF6 (T27N),inhibits VEGF-stimulated GTP-loading
of Rac1, ROS production, and VEGFR2 autophosphorylation. These results suggest
that ARF6 is an upstream mediator for Rac1, which may contribute to ROS production
that mediates VEGFR2 autophosphorylation in ECs (Fig. 1).""" ;
    prov:wasQuotedFrom pubmed:17511588 .
  sub:_7 rdfs:comment "support@belframework.org" ;
    rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:17511588 ;
    prov:wasDerivedFrom beldoc: , sub:_6 .
}