sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_4 ;
    pav:version "1.4" .
  sub:_3 prov:value "MC were stimulated with IL-1{beta} (0.3 nM) and, to block endogenous NO formation, additionally with different concentrations of the NOS inhibitor L-NMMA for 24 h. In IL-1{beta}–treated MC, PDGFR{alpha} expression and NO formation (measured as nitrite, data not shown) was drastically increased (Figure 6). Co-administration of L-NMMA reduced IL-1{beta}–evoked PDGFR{alpha} expression in part (Figure 6) and blunted IL-1{beta}–induced NO formation (data not shown). We conclude from these data that endogenously produced NO at least partially triggers IL-1{beta}–driven PDGFR{alpha} expression." ;
    prov:wasQuotedFrom pubmed:15872077 .
  sub:_4 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:15872077 ;
    prov:wasDerivedFrom beldoc: , sub:_3 .
}