@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix RNA: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:11621; a Protein: . sub:_2 geneProductOf: hgnc:2328; a RNA: . sub:_3 occursIn: species:9606; rdf:object sub:_2; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:HNF1A) -| r(HGNC:CPT1A)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "1.4" . sub:_4 prov:value "It is also unlikely that a defect in mitochondrial fatty acid import, mediated by L -CPT1, resulted in the differences between peroxisomal and mitochondrial eta-oxidation, because the expression of the gene for L -CPT1 was unaffected by HNF1alpha gene inactivation."; prov:wasQuotedFrom pubmed:10852923 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:10852923; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:29:51.312+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }