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kin(p(HGNC:PRKCZ)) -> tscript(p(HGNC:MEF2C))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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# Ariadne: Results (Fig. 9 A) show that the presence of etaPKC triggers the activation of ERK5 to phosphorylate MEF2C in a manner that is dependent on MEK5. [Regulation] Consistent with the role of the aPKCs in the MEK5-ERK5 pathway, we show that zetaPKC and lambda/iotaPKC activate the Jun promoter through the MEF2C element, a well-established target of ERK5. From all these results, we conclude that MEK5 is a critical target of the aPKCs during mitogenic signaling.
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Selventa
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2014-07-03T14:29:53.978+02:00
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