sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_7 ;
    pav:version "1.4" .
  sub:_6 prov:value "vitamin D receptor (VDR) is a nuclear receptor that functions as a ligand-activated transcription factor, whereas the hairless gene product (Hr) acts as a corepressor of both the thyroid hormone receptor (TR) and the orphan nuclear receptor, RORalpha. In the present study, we show that VDR-mediated transactivation is strikingly inhibited by co-expression of rat Hr. The repressive effect of Hr is observed on both synthetic and naturally-occurring VDR-responsive promoters, and also when VDR-mediated transactivation is augmented by overexpression of its heterodimeric partner, retinoid X receptor....Coimmunoprecipitation data demonstrate that Hr and VDR associate in a cellular milieu, suggesting in vivo interaction. " ;
    prov:wasQuotedFrom pubmed:12847098 .
  sub:_7 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:12847098 ;
    prov:wasDerivedFrom beldoc: , sub:_6 .
}