@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix RNA: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:109619; a Protein: . sub:_2 geneProductOf: mgi:96919; a RNA: . sub:_3 occursIn: species:10090; rdf:object sub:_2; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(MGI:Neurog2) -| r(MGI:Ascl1)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "1.4" . sub:_4 prov:value "An up-regulation of Mash1 (Ascl1) expression was observed throughout the dorsomedial cortical ventral zone of Ngn2 mutant embryos at E12.5 and E13.5. This up-regulation was less apparent in the lateral ventral zone in which Ngn1 expression was maintained in Ngn2 mutants. Mash1 up-regulation was strikingly enhanced in the dorsal telencephalon and dorsal thalamus of Ngn1 and Ngn2 double mutant embryos. These results suggest that the Ngns function in a partially redundant manner to restrict Mash1 expression to limited domains of the forebrain neuroepithelium."; prov:wasQuotedFrom pubmed:10640277 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:10640277; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:29:49.751+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }