@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix go: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:7670; a Protein: . sub:_2 occursIn: species:9606; rdf:object go:0048590; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:NCOA3) -> bp(GO:growth)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "1.4" . sub:_3 prov:value "In the meantime, the expression of SRC-2 mRNA was elevated in SRC-1 null mice, suggesting that SRC-2 can partially compensate for SRC-1 function SRC-3 is the most distinct among the three members. It coactivates not only the nuclear receptors, but also other unrelated transcription factors such as those in the cAMP or cytokine pathways [15]. Compared with the widespread expression of SRC-1 and SRC-2, expression of SRC-3 is restricted to the mammary gland and several other tissues [16]. Disruption of the SRC-3 gene in mice causes severe growth and reproductive defects, including the retardation of mammary gland development"; prov:wasQuotedFrom pubmed:12223121 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12223121; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:05.654+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }