@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix go: . @prefix Protein: . @prefix pfr: . @prefix geneProductOf: . @prefix hasAgent: . @prefix rgd: . @prefix mesh: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 hasAgent: sub:_2; a go:0016301 . sub:_2 geneProductOf: pfr:Akt%20Family; a Protein: . sub:_3 geneProductOf: rgd:621506; a Protein: . sub:_4 occursIn: mesh:D004730, species:10116; rdf:object sub:_3; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "kin(p(PFR:\"Akt Family\")) -| p(RGD:Mapk8)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_6; pav:version "1.4" . sub:_5 prov:value "In cells stably transfected with empty vector, AVP rapidly stimulated ERK, JNK, and p38 activity (Fig. 7A), similar to what we have previously reported (11). However, in both clones stably expressing myr-Akt, AVP-induced activation of JNK was completely blocked, and activation of p38 MAP kinases was significantly reduced (Fig. 7A)."; prov:wasQuotedFrom pubmed:12882977 . sub:_6 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12882977; prov:wasDerivedFrom beldoc:, sub:_5 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:15.043+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }