@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix go: . @prefix hasAgent: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:3665; a Protein: . sub:_2 hasAgent: sub:_3; a go:0003824 . sub:_3 geneProductOf: hgnc:9052; a Protein: . sub:_4 occursIn: species:9606; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:FGF1) -> cat(p(HGNC:PLAU))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_6; pav:version "1.4" . sub:_5 prov:value "Also, FGFs modulate the expression of uPA receptor on the endothelial cell surface, thus allowing the localization of the proteolytic activity at the leading edge of the cell at the front of migration [22]. Furthermore, FGF1 and FGF2 induce the expression of the plasminogen activator inhibitor (PAI)-1 in cultured endothelial cells leading to a fine modulation of the proteolytic balance [23-25]."; prov:wasQuotedFrom pubmed:15863032 . sub:_6 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:15863032; prov:wasDerivedFrom beldoc:, sub:_5 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:34.753+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }