sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_6 ;
    pav:version "1.4" .
  sub:_5 prov:value "In vivo, endogenous FAP68 can be coimmunoprecipitated with the HGF receptor in the absence of stimuli and not upon HGF stimulation. Thus, FAP68 represents a novel type of effector that interacts with the inactive HGF receptor and is released upon receptor phosphorylation. # Ariadne: Significantly, nonphosphorylated HGF receptor prevents FAP68 from stimulating  p70S6K . [Regulation] # Ariadne: FAP68  interacts specifically with the inactive form of HGF receptor, such as a kinase- defective receptor or a dephosphorylated wild type receptor. [Binding] # Ariadne: Significantly, nonphosphorylated HGF receptor prevents FAP68 from stimulating  p70S6K . [Regulation]" ;
    prov:wasQuotedFrom pubmed:11571281 .
  sub:_6 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:11571281 ;
    prov:wasDerivedFrom beldoc: , sub:_5 .
}