. . . . . . . "[Our findings suggest that GCN2 may be an important eIF2? kinase under the physiological condition, whereas blocking the GCN2 pathway under exposure to significant ?-amyloidosis rather aggravates eIF2? phosphorylation leading to BACE1 and ATF4 elevations in AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en . . . . . "2017-02-19"^^ . . "Gene-disease associations inferred from text-mining the literature."@en . "DisGeNET evidence - LITERATURE"@en . "2017-10-17T13:10:46+02:00"^^ . . . . . . . . . . . "v5.0.0.0" . "v5.0.0" .