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[Since apoptosis contributes to the neuronal loss associated with amyloid ? peptide (A?) toxicity in AD, we investigated the protective effects of HO-1 and CO against A?(1-42) toxicity in SH-SY5Y cells, employing cells stably transfected with empty vector or expressing the cellular prion protein, PrP(c), and rat primary hippocampal neurons.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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