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p(HGNC:EGF) -> complex(p(HGNC:ERBB3),p(HGNC:PIK3R1))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Since EGF-induced erbB3 phosphorylation can mediate recruitment of the regulatory p85 subunit of phosphoinositide 3-kinase (PI 3-kinase) (Fedi et al., 1994; Kim et al., 1994; Prigent and Gullick, 1994; Soltoff et al., 1994), erbB3 immunoprecipitates were probed for the presence of p85. The amount of p85 recovered paralleled the degree of phosphorylation of erbB3, in that quiescent BRK-expressing cells had more p85 associated with erbB3 than control cells, and EGF-stimulated BRK-expressing cells had a substantially enhanced recruitment of p85 to erbB3-containing complexes when compared with stimulated control cells (Figure 2).
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Selventa
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2014-07-03T14:29:53.594+02:00
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