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http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RAs-X21hzB4CGnpvkN8MlzuIuYGWXongsEEjOsFuLrUI0#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11103
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http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://semanticscience.org/resource/SIO_000139
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0042789
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p(HGNC:SMARCB1) -> tscript(p(HGNC:RB1))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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Mammalian SNF5 is essential for normal cell viability, and loss or mutation of the human SNF gene is the molecular basis for familial malignant rhabdoid tumorigenesis. Previous studies have suggested that SNF5 suppresses cancer by signaling through the p16Ink4a and retinoblastoma tumor suppressors to negatively regulate cell cycle progression from G0/G1 into S phase.
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Selventa
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2014-07-03T14:30:34.143+02:00
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