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http://www.tkuhn.ch/bel2nanopub/RAspsMzuGgerLk1cpsfAX7mgTZ00D6f0wW_lf4JxdHrq0#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.informatics.jax.org/marker/MGI:104798
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http://semanticscience.org/resource/SIO_000139
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0003824
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p(MGI:Tnf) -> cat(p(MGI:Casp3))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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a dominant-negative mutant of nuclear factor-kappaB-inducing kinase also promoted TNFalpha- and Fas-mediated apoptosis. Expression of either crmA or a dominant-negative mutant of the Fas-associated death domain protein prevented TNFalpha- and Fas-mediated apoptosis. In addition, the caspase inhibitors, DEVD-cho and IETD-fmk, inhibited TNFalpha- and Fas-mediated apoptosis. In Ad5IkappaB-infected hepatocytes, caspases-3 and -8 were activated within 2 h after treatment with anti-Fas antibody or within 6 h after TNFalpha treatment
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Selventa
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2014-07-03T14:29:50.733+02:00
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